The human ΔNp53 isoform triggers metabolic and gene expression changes that activate mTOR and alter mitochondrial function
Journal Article
Overview
publication date
- October 1, 2013
has subject area
- Biochemical Phenomena - Amino Acid Sequence
- Biology - Epigenomics
- Carrier Proteins - Tumor Suppressor Protein p53
- DNA-Binding Proteins - Tumor Suppressor Protein p53
- Environment and Public Health - Age Factors
- Gene Expression Regulation
- Genes - Genes, p53
- Genes, Neoplasm - Genes, p53
- Genetics - Epigenomics
- Glutathione
- Health Care Quality, Access, and Evaluation - Age Factors
- Humans
- Molecular Sequence Data
- Molecular Sequence Data - Amino Acid Sequence
- Nuclear Proteins - Tumor Suppressor Protein p53
- Organelles - Mitochondria
- Phosphoproteins - Tumor Suppressor Protein p53
- Protein Isoforms
- Protein Structure, Tertiary
- Protein-Serine-Threonine Kinases - TOR Serine-Threonine Kinases
- Proteins - TOR Serine-Threonine Kinases
- Subcellular Fractions - Mitochondria
- Tumor Suppressor Proteins - Tumor Suppressor Protein p53
has restriction
- hybrid
Date in CU Experts
- September 3, 2013 2:59 AM
Full Author List
- Lin S-C; Karoly ED; Taatjes DJ
author count
- 3
citation count
- 13
published in
- Aging Cell Journal
Other Profiles
International Standard Serial Number (ISSN)
- 1474-9718
Electronic International Standard Serial Number (EISSN)
- 1474-9726
Digital Object Identifier (DOI)
Additional Document Info
start page
- 863
end page
- 872
volume
- 12
issue
- 5